Mutations in the neuronal b-tubulin subunit TUBB3 result in malformation of cortical development and neuronal migration defects

نویسندگان

  • Karine Poirier
  • Yoann Saillour
  • Nadia Bahi-Buisson
  • Xavier H. Jaglin
  • Catherine Fallet-Bianco
  • Rima Nabbout
  • Laetitia Castelnau-Ptakhine
  • Agathe Roubertie
  • Jamel Chelly
چکیده

result in malformation of cortical development and neuronal migration defects Karine Poirier1,2, Yoann Saillour1,2,, Nadia Bahi-Buisson1,2,3,, Xavier H. Jaglin1,2, Catherine Fallet-Bianco7,8, Rima Nabbout3,9, Laetitia Castelnau-Ptakhine1,2, Agathe Roubertie10, Tania Attie-Bitach4, Isabelle Desguerre1,2,3, David Genevieve4, Christine Barnerias4, Boris Keren11, Nicolas Lebrun1,2, Nathalie Boddaert5, Féréchté Encha-Razavi6 and Jamel Chelly1,2,∗

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Mutations in the neuronal ß-tubulin subunit TUBB3 result in malformation of cortical development and neuronal migration defects.

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Jcb_201705172 1..3

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Phenotypic spectrum of the tubulin-related disorders and functional implications of disease-causing mutations.

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MicroTUB(B3)ules and Brain Development

The microtubule network is crucial for the developing nervous system, and mutations in tubulin-encoding genes disrupt neuronal migration. Tischfield et al. (2010) now report that mutations in the tubulin-encoding gene TUBB3 have a striking impact on microtubule dynamics in neurons, resulting in a diverse set of disease symptoms.

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تاریخ انتشار 2010